LncRNA Growth Arrest Specific 5 Promotes Glucose Metabolism Reprogramming Via the IGF2BP1/SIX1 Axis and Inhibits Ferroptosis of Endothelial Progenitor Cells Via the miR-23a-3p/SLC7A11 Axis in Coronary Heart Disease.

erastin - Whitehead Institute for Biomedical Research, Dana-Farber Cancer Institute, Columbia University, Prolexys

LncRNA Growth Arrest Specific 5 Promotes Glucose Metabolism Reprogramming Via the IGF2BP1/SIX1 Axis and Inhibits Ferroptosis of Endothelial Progenitor Cells Via the miR-23a-3p/SLC7A11 Axis in Coronary Heart Disease. (PubMed, Anatol J Cardiol) - Mar 10, 2025 - "Growth arrest specific 5 promotes glucose metabolic reprogramming and represses ferroptosis of EPCs via the IGF2BP1/SIX1 and miR-23a-3p/SLC7A11 dual-regulatory pathways in CHD."
Journal • Cardiovascular • Coronary Artery Disease • Heart Failure • GAS5 • IGF2BP1 • MIR23A • SIX1 • SLC7A11

http://www.ncbi.nlm.nih.gov/pubmed/40062373

Biao Tang; Ming Zhong; Qiang Zhao; Wenxia Xu; Yinfeng Liu; Zenan Jiang

Mar 10, 2025